Trang Nhà Khăn Vàng 973,Kính chuyễn bài sưu tầm
về cách chữa trị Bệnh gout bằng phương thuốc Dân gian
qua bài chuyễn và sưu tầm từ Huynh đệ Lê Khôi
và bài viết của James Mcintosk từ Medical News Today
Cầu xin Thượng đế ban nhiều ơn lành cho
những ai đang còn tìm thuốc chữa căn bệnh Gout
Trang Nhà Khăn Vàng 973
Bài thuốc đơn giản trị Gout dứt điểm từ 2 món thực phẩm dễ kiếm
Lần thứ nhất trong đời mới thấy tận mắt bệnh gout. Thật khủng khiếp, hai bàn tay của anh T trông giống như nải chuối mật mốc chín muồi. vỏ đen, năm khớp xương trên mu hai bàn tay và các khớp của các ngón tay, nó ( U) lên giống như các mụt nhọt bóng lưởng, các ngon tay sưng no tròn cứ tưởng tượng nó sắp bung nở, màu tím bầm đen. Hai bàn chân sưng tấy , tím ngắt , thấy thật dễ sợ .
Anh cho biết đã uống hằng chục bài thuốc lượm lặt trên net, khi chịu hết nổi thì đến BS, BS chích thuốc trực tiếp vào các khớp bị sưng.5 Tri gout Đỡ một vài tháng rồi tái lại, cứ như vậy kéo dài mải, nhưng năm nay thì quá nặng như các anh đang thấy .
Vừa rồi anh T tươi rói đến thăm cho tôi mấy gói cà phê p, anh rất vui báo cáo hơn 6 tháng nay bệnh của anh nằm im, không nghe tái tại, còn lâu dài thì chưa biết ra sao, nhưng người cho bài thuốc nầy nói thĩnh thoảng uống nó sẽ chấm dứt không tái lại nửa vì người nhà của họ đã trị dứt mấy năm nay không tái lại (cũng có thể tùy thể trạng của người bệnh hạp chăng) bài thuốc chẵng có chi đặc biệt chỉ có 2 món :
1/- 1 kg trái (khổ hoa) mướp đắng. 2/- 0.800 gram trái bưởi.
Cả hai để nguyên vỏ ruột, hột, không bỏ thứ gì. vằm chung lại cho vào nồi nầu, nước đổ vừa xấp trên mặt, nấu cạn còn lại 1/2 đổ ra ly. Nấu lại lần thứ 2 cũng như vậy, hai lần hòa chung cho vô tủ lạnh uống như nước trà, cố gắng uống trong ngày cho hết, rồi mai làm lại như vậy, khoảng 3 bữa là xẹp không đau nhức, rồi làm tiếp lâu mau tùy mình (Đã 6 tháng không tái lại). Xin quý huynh ghi lại để giúp người Huynh Trường Lê Khôi Chuyển
Gout is a condition that was first identified as early as 2640 BC by the ancient Egyptians. Hippocrates, the legendary Greek physician, referred to gout as "unwalkable disease" in 5th century BC, and observed links between the condition and certain lifestyle areas.1
Even so, gout remains a major and growing public health problem, and one that can be extremely painful to those unfortunate enough to suffer from it.
"I've been shot, beat up, stabbed and thrown out of a helicopter, but none of that compared to the gout," said one veteran visiting a VA Hospital in Birmingham, AL. This complex form of arthritis is characterized by the onset of sudden and severe pains. Fortunately, it can be treated, and there are preventative measures that can be taken to reduce its risk.
What is gout?
What causes gout?
Signs and symptoms
Tests and diagnosis
Treatment and prevention
You will also see introductions at the end of some sections to any recent developments that have been covered by MNT's news stories. Also look out for links to information about related conditions.
Fast facts on gout
Here are some key points about gout. More detail and supporting information is in the main article.
Gout is a form of arthritis.
Gout most commonly affects the joint in the base of the big toe.
Gout is caused by an excess of uric acid in the bloodstream.
Gout attacks often occur without warning in the middle of the night.
There are four stages of gout varying in clinical severity.
Not all people with hyperuricemia develop gout, and not all gout attacks occur when a person has hyperuricemia.
Advanced cases of gout can lead to the formation of kidney stone
Doctors often use joint fluid tests when testing for gout.
Most gout cases are treated with specific medication.
Avoiding foods with a high purine content is a good preventative measure to take to avoid gout.
What is gout?
Gout is a common form of inflammatory arthritis - a condition affecting the joints and musculoskeletal system. It is the most common form of inflammatory arthritis in men, and although it is more likely to affect men, women become more susceptible to it after the menopause.
Gout commonly affects the base of the big toe. When affecting this area, the condition can also be referred to as podagra.
The condition is characterized by sudden and severe pains, redness and tenderness in the joints, most commonly in the base of the big toe. When affecting the big toe, gout can also be referred to as podagra.
These symptoms occur when uric acid, a bodily waste produce, is deposited in the form of needle-like crystals in tissues and fluids within the body. Chalky deposits of uric acid known as tophi can also form as lumps under the skin surrounding the joints. Uric acid crystals can also collect in the kidneys, sometimes resulting in kidney stones.
At its most disabling, gout can cause permanent damage to joints and the kidneys. It normally takes a long period, around 10 years, without any proper treatment for the disease to reach this advanced stage, however.
The Centers for Disease Control and Prevention (CDC) report that 2.6 million Americans were affected by gout in 2005 and that this figure is projected to rise to 3.6 million by 2025.1
What causes gout?
Gout is caused initially by an excess of uric acid in the blood (hyperuricemia). Uric acid is produced in the body through the breakdown of purines - specific chemical compounds that are found in certain foods such as meat, poultry and seafood.
Normally, uric acid dissolves in the blood and is excreted from the body in urine via the kidneys. If too much uric acid is produced or not enough is excreted then it can build up and form the needle-like crystals that cause inflammation and pain in the joints and surrounding tissue.
There are a number of factors that can increase the likelihood of hyperuricemia, and therefore gout:
Age and gender: men produce more uric acid than women, though women's levels of uric acid approach those of men after the menopause
Genetics: a family history of gout increases the likelihood of the condition developing
Lifestyle choices: alcohol consumption interferes with the removal of uric acid from the body. Eating a high-purine diet also increases the amount of uric acid in the body
Lead exposure: chronic lead exposure has been linked in some cases to gout
Medications: certain medications can increase the levels of uric acid in the body, such as diuretics and drugs containing salicylate
Weight: being overweight increases the risk as there is more tissue in the body for turnover or breakdown, leading to the production of excess uric acid
Other health problems: if the kidneys are unable to eliminate waste products adequately (renal insufficiency) then uric acid levels can remain high. Other conditions that can contribute are high blood pressure (hypertension), diabetes and hypothyroidism.
Recent developments on gout causes from MNT news
Gout runs strongly in families, suggests large-scale study
Researchers from the UK's University of Nottingham and colleagues conducted a new study of the population of Taiwan, where there is a high rate of gout, and found that the condition clusters in families.
Gout risk from purine rich foods
Foods rich in purines, particularly those found in meat and seafood, quintuple the immediate risk of a gout flare-up, according to research published online in the Annals of the Rheumatic Diseases.
Signs and symptoms
Gout usually becomes symptomatic suddenly without warning, often in the middle of the night. The main symptoms are intense joint pain that subsides to discomfort, inflammation and redness. Gout frequently affects the large joint of the big toe, but can also commonly affect the ankles, knees, elbows, wrists and fingers.
There are four stages through which gout progresses. Each is characterized by its symptoms (or lack of).
Gout patients often suffer from acute inflammation around their joints.
It is possible for a person to have hyperuricemia without any outward symptoms. At this stage, treatment is not required, though urate crystals are being deposited in tissue and causing slight damage.
This stage occurs when the urate crystals that have been deposited suddenly cause acute inflammation and intense pain. This sudden attack is referred to as a "flare" and will normally subside within 3-10 days. Flares can sometimes be triggered by stressful events, alcohol and drugs.
Interval or inter critical gout
This stage is the period in between attacks of acute gout. Subsequent flares may not occur for months or years, though if not treated over time they can last longer and occur more frequently. During this time, further urate crystals are being deposited in tissue.
Chronic tophaceous gout
This final stage is the most debilitating form of the disease. Permanent damage may have been dealt to joints and the kidneys. The patient can suffer from chronic arthritis and develop tophi - big lumps of urate crystals - in cooler areas of the body such as the joints of the fingers.
It takes a long time without treatment to reach the stage of chronic tophaceous gout, around 10 years. It is very unlikely that a patient receiving proper treatment would progress to this stage.
Tests and diagnosis
Gout can be tricky to diagnose as its symptoms, when they do manifest, are similar to those of other conditions. While hyperuricemia occurs in the majority of people that develop gout, it may not be present during a flare. On top of that, the majority of people with hyperuricemia do not develop gout.
One diagnostic test that doctors can carry out is the joint fluid test, where fluid is extracted from the affected joint with a needle. The fluid is then examined to see if any urate crystals are present.
As joint infections can also cause similar symptoms to gout, a doctor can look for bacteria when carrying out a joint fluid test in order to rule this condition out as well.
Doctors can also conduct a blood test to measure the levels of uric acid in the blood, but as specified, people with high uric acid levels do not always experience gout. Equally, some people can develop the symptoms of gout without having increased levels of uric acid in the blood.
Finally, doctors can search for urate crystals around joints or within a tophus.
One condition that can often be confused with gout is pseudogout. The symptoms of pseudogout are very similar to those of gout, the difference being that the joints are irritated by calcium phosphate crystals rather than urate crystals. This means that pseudogout requires different treatment to gout.
Treatment and prevention
The majority of gout cases are treated with medication. Medication can be used to treat the symptoms of gout attacks, prevent future flares and reduce the risk of gout complications such as kidney stones and the development of tophi.
Commonly used medications are nonsteroidal anti-inflammatory drugs (NSAIDs), colchicine or corticosteroids. These reduce inflammation and pain in the areas affected by gout and are commonly administered orally.
It is recommended that you drink between 2 and 4 liters of water a day to reduce the risk of gout.
Medications can also be used to either reduce the production of uric acid (xanthine oxidase inhibitors such as allopurinol) or improve the kidney's ability to remove uric acid from the body (probenecid).
There are many lifestyle and dietary guidelines that can be followed to protect against future flares or prevent gout from occurring in the first instance:
Maintain a high fluid intake (2-4 liters a day)
Maintain a healthy body weight
Eat a balanced diet
Limit fish, meat and poultry intake.
If attempting to lose weight, avoid low-carbohydrate diets. If carbohydrate intake is insufficient, the body is unable to burn its own fat properly, releasing substances called ketones into the bloodstream. This results in a condition called ketosis that can increase the level of uric acid in the blood.
It is most important to avoid foods that are high in purines, to ensure that the levels of uric acid in the blood do not get too high. Here is a list of high-purine foods to be wary of:
Dried beans and peas
Recent developments on gout treatment from MNT news
NSAIDs may halve breast cancer recurrence in overweight women
A new study by researchers at the University of Texas in Austin finds that recurrence of hormone-related breast cancer was reduced by more than half in overweight and obese women who regularly used nonsteroidal anti-inflammatory drugs.
'Increased risk of venous thromboembolism among NSAID users'
Users of non-steroidal anti-inflammatory drugs are at increased risk of venous thromboembolism, according to a new study published in the journal Rheumatology.
Uric acid's role in gout has been clearly defined and understood. As a result of this and the wide availability of relevant medications, gout is a very controllable form of arthritis. Written by James McIntosh Medical News Today